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Across several preclinical models, Epitalon demonstrated a reduction in chromosomal aberrations, i.e., those misalignments, breaks, and translocations that set the stage for malignant transformation

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The altered RA-FLSs assume a pivotal role in the development and advancement of RA, exerting a distinctive phenotype marked by diminished susceptibility to cell apoptosis, heightened expression of adhesion molecules, and aberrant generation of cytokines, chemokines, and MMPs (27, 28)

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