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Its working mechanism is allosteric facilitation of HGF/c-Met signaling: Dihexa binds HGF with high affinity (reported K d ~65 pM in the original work) and is proposed to potentiate HGF dimerization and c-Met phosphorylation at subthreshold endogenous HGF concentrations

Lavie Jr., MD, FACC, FACP, FCCP, professor of medicine, medical director of cardiac rehabilitation and preventive cardiology, director of the Exercise Testing Laboratory and staff cardiologist in the Echocardiographic Laboratory at the John Ochsner Heart and Vascular Institute, Ochsner Clinical School-The University of Queensland School of Medicine, told Cardiology Today

doi: 10.1016/j.immuni.2009.02.007 31 BasuRHattonRDWeaverCT

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