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As a result, current understanding is based primarily on experimental and early-stage research rather than large-scale clinical evidence

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Excessive ROS levels can induce cellular damage, initiating a cascade of effects that potentially culminate in tissue and organ injury (30)

The enrichment of platelet homeostasis pathways suggests a potential link between metabolic dysregulation and platelet function in the context of HF, highlighting the importance of understanding platelet biology in CV pathophysiology

On the mechanism of cytoprotection by ferrostatin-1 and liproxstatin-1 and the role of lipid peroxidation in ferroptotic cell death