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Proposed mechanisms include upregulation of growth-hormone-receptor expression in injured tissue, VEGF-driven angiogenesis, modulation of the nitric-oxide pathway in vascular endothelium, and activation of FAK-paxillin signaling

Phosphatidylcholine is particularly important for facilitating the appropriate secretion of very low-density lipoproteins from hepatocytes (Gibellini and Smith, 2010), essential for preventing the accumulation of lipid droplets in hepatic tissue, which can ultimately lead to steatosis ( de novo synthesis is possible through the choline pathway (considered the main pathway), but it can also occur via SAM ( Figure 1 )