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Likewise, a balanced ROS production and ROS-dependent signaling seem to be favorable for normal growth behavior and cellular differentiation

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Using the monoiodoacetate (MIA) rat model of inflammatory OA, Que and colleagues demonstrated that GLP-1R activation triggered the protein kinase A/cAMP-response element binding protein (PKA/CREB) signalling pathway, thus reducing inflammation in cartilage [36]

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doi: 10.1093/neuonc/noab106