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FAE mediate their anti-psoriatic effects via activation of several cellular pathways that induce T-cell apoptosis or prevent release of proinflammatory cytokines (Figure 1)

In prostate and bladder cancer, the Gln metabolism antagonist JHU083 inhibits TAM Gln metabolism, resulting in increased glycolysis, disruption of the tricarboxylic acid (TCA) cycle, and impairment of purine metabolism

Novel missense mutation in VPS33B is associated with isolated low gamma-glutamyltransferase cholestasis: attenuated, incomplete phenotype of arthrogryposis, renal dysfunction, and cholestasis syndrome

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