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Intracellular iron overload and accumulation of lipid peroxides are the characteristics of ferroptosis, so ferroptosis is considered as one of the potential mechanisms of delayed wound healing in diabetics [136, 137]
MASH has replaced the old terminology of NASH (non-alcoholic steatohepatitis) and is defined as the presence of 5 % hepato-steatosis and hepatocyte "ballooning" (inflammation with hepatocyte injury and cell liver death) with or without fibrosis [5,6] [Table 2]
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Bone marrow-derived monocytes give rise to self-renewing and fullydifferentiated Kupffer cells