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Early animal experiments with hypothalamic lesions and post-mortem examinations in humans with morbid obesity led to a proposal of the dual center hypothesis, postulating that ventromedial nuclei (VMN) serve as the satiety center and the lateral hypothalamic area (LHA) - as the feeding or hunger center (4) that when stimulated results in hyperphagia and subsequently hypothalamic obesity (5, 6)

Interestingly, lipid metabolism pathways, such as the phospholipase D signaling pathway and PPAR signaling pathway, were significantly enriched ( Figure 4D )

Alzheimers disease is not simply a memory disease. It is a complex neurodegenerative process that often overlaps with insulin resistance, vascular disease, inflammation, mitochondrial stress, sleep disruption, and loss of metabolic resilience

JAMA Neurology, 80(7), 730738

Read more: Ozempic, the miracle drug, and the harmful idea of a future without fat As a family physician with a PhD in nutrition, I translate the latest nutrition science into dietary recommendations for my patients

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