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Staresinic et al

Trim29 interacts with IB (NF-B inhibitory protein), mediating its ubiquitination-dependent degradation, which results in NF-B activation

In another study, recombinant efgartigimod was found to stabilize keratinocyte monolayers in a cellular model of pemphigus, hinting at a direct role of FcRn in mediating pathological effects of anti-DsG Abs [236]