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[100] have suggested that inhibition of myocardial CPT I activity may be a common feature of systemic inflammation, or of inflammation localized to the heart

Furthermore, the hyperthyroidism-related myocardial injury was proven histologically in our study by disturbed histoarchitecture, increased collagen deposition, severe inflammation, vascular congestion, and increased diameter of the myocardial fibers

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Therefore, upregulation or gain in GABA A receptor function could be a potential therapeutic target in rescuing ASD phenotypes

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