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To start with, its a more stable form of glutathione, meaning that it is less prone to break down quickly in the body, and more likely to be bioavailable- meaning that it will be both absorbed more readily and be made use of by the body

This deficiency specifically impairs mitochondrial Fe-S cluster proteins, leading to disruptions in electron transport chain activity, cellular energy metabolism, and cell cycle progression, ultimately resulting in growth inhibition and apoptosis