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They demonstrated that cardiac pathogenic effects were exacerbated by a Zn deficiency and alleviated by its supplementation, which suggests that Zn plays a critical role in stimulating MTs to suppress the oxidative stress-activated BCL10/CARD9/p38 MAPK pathway [80]

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The dose ranges studied spanned from 0.25 mg to 54 mg, allowing for assessment of dose-response relationships in adverse effect profiles

Second, the roles of emerging post-translational modifications in mediating this interaction are not fully integrated into current models