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Thus, regardless of our understanding of the physiology or pathophysiology of islet produced GLP-1, these data suggest that this pool of GLP-1 could be targeted to treat T2DM and avoid some of the CNS side-effects (i.e., nausea) associated with long-acting GLP-1 agonists
The starting rung The initial step establishes tolerance rather than targeting a clinical endpoint, which is why it is the same entry point regardless of goal
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reproducible GMP-grade batches across 50% and 90% concentrations
This fusion protein is capable of exerting the beneficial effects of GLP-1 on blood glucose regulation and insulin secretion while also targeting and inhibiting GIPR function through the antibody fragment
