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10.1007/s12011-020-02241-5 Biol Trace Elem Res

A previous study supported this, showing that treatment of SLC1A5 -knockdown HNSCC cell lines with V-9302 led to marked inhibition of glutamine metabolism, thereby suppressing growth and proliferation both in vitro and in vivo 97

19 Both inputs were retained from the original CEA model for consistency

The active forms are potent stimulators of glucose-dependent insulin secretion

GSSG is then recycled back to two GSH molecules through accepting electrons from NADPH via the enzyme glutathione reductase (vitamin B2 as FAD dependent)

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