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In the acute phase, the neuroinflammatory response worsens BBB injury by degrading tight junctions and the extracellular matrix, promoting neuroglial proliferation, and activating cardiomyocytes, thereby further increasing its permeability

Kim EY, Schrader N, Smolinsky B, Bedet C, Vannier C, Schwarz G, et al

10.1038/s41419-019-1542-0 84

It also has a tyrosine residue to replace the first amino acid at the N-terminus, which researchers consider to help increase the stability of the peptide