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Coronary atherosclerosis initiates with endothelial damage to the vascular intima, followed by a sequence of events including lipid deposition, vascular smooth muscle cell proliferation, and extracellular matrix synthesis, collectively resulting in arterial wall thickening and progressive luminal narrowing (17)

Among the antioxidant molecules of low molecular weight are vitamins E and C, obtained from the diet

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Studies demonstrated that NO confers SAR through the downstream regulation of ROS-AzA-G3P, which functioned parallel to SA-mediated events [168]

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