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Galanin inhibits GLP1 and GIP secretion via the GAL(1) receptor in enteroendocrine L and K cells

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Collectively, these findings suggest that modulating key factors within the HIF-1 signaling pathway, both upstream and downstream, can effectively optimize effector cell metabolism, diminish the proliferation and overactivation of abnormal, self-reactive cells, and subsequently ameliorate tissue inflammation in SLE patients, particularly those with LN