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Systemic organ involvement can occur in SJS/TEN through a variety of mechanisms

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Elevated interleukin-6 (IL-6) levels further intensify inflammation and oxidative stress, disrupting insulin signaling and worsening complications such as nephropathy, retinopathy, and neuropathy

Long-term iodine exposure leads to increased iodination of thyroglobulin, which increases its antigenicity and initiates the autoimmune process in genetically susceptible individuals

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843 Coexpression of PyrH and a flavin reductase partner RebF with the brassinin pathway in N