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The less extended ester group, whose oxygen atoms interact via hydrogen bridges with the amino acid tyrosine (Tyr-1169) and serine (Ser-1131), is also responsible for this binding 84,85

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Therefore, it's unclear if the improvements were due to BPC 157 or simply the passage of time

Cancer cells that were ER-positive and resistant to tamoxifen were more likely to undergo cell death when treated with a combination of luteolin and inhibitors of PI3K, AKT, or mTOR (57)

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