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We demonstrate mechanistically the need for increased mitochondrial function, facilitated by mTORPGC1-mediated mitochondrial biogenesis, to support rebalancing of pyrimidines via UCK2 to support cellular survival in the face of continued 5FU treatment

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Hyperandrogenemia and insulin resistance: the chief culprit of polycystic ovary syndrome

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Zigra PI, Maipa VE, Alamanos YP