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The effect on motility is in accordance with the studies in animals (both dietary or selenoproteins knockout interventions), which have shown defects in mitochondria, midpiece, and tails of spermatozoa, resulting in lower motility, viability, and fertilizing ability (see Sections 2 and 4)

However, in vivo studies yielded conflicting results, with eosinophils appearing to play protective (da Silva Marques et al, 2021

Bedaiwy MA, Falcone T

American Journal of Kidney Diseases (2003)

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