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Abnormal fetomaternal glucocorticoid metabolism in the background of premature delivery: placental expression patterns of the 11-hydroxysteroid dehydrogenase 2 gene

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Kim, Lee, Kim, Cheon, and Lim: Successful rapid weight reduction and the use of liraglutide for morbid obesity in adolescent Prader-Willi syndrome Abstract Prader-Willi syndrome (PWS), an imprinting disorder, results from the loss of expression of a paternal gene on chromosome 15q11-q13

Given that -synuclein aggregation plays such a central role in PD pathology, any intervention that can reduce or prevent this process offers a potential disease-modifying treatment

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