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In ex vivo studies using isolated blood vessels, BPC-157 induces vasodilation through increased nitric oxide production, with effects blocked by endothelium removal or eNOS inhibition

ROS production was high in GSH-depleted cells even after NAC treatment suggesting recovery in membrane potential after NAC treatment may partially be independent of ROS production and GSH pool in mitochondria

Neurometabolic indicators of mitochondrial dysfunction in repetitive mild traumatic brain injury

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Ensuring adequate intake and timely diagnosis and treatment of B vitamin deficiencies can mitigate their neuropsychiatric effects and improve patient outcomes, highlighting the critical role of these vitamins in maintaining neurological health