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This immune privilege is attributed to their low expression of major histocompatibility complex class II molecules (MHC-II, specifically HLA-DR) and the absence of co-stimulatory molecules (CD80/CD86), supporting their capacity for immune evasion (Khoury et al., 2014)

Diabetes Care 26:15891596

Figure 2 In a proteomic study, cotyledons of two B

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Gene therapies to upregulate specific isoforms and biomarkers for early detection are also on the horizon, aiming for personalized interventions