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These results support the notion that 12/15-LOX can directly contribute to atherosclerosis via LDL oxidation

Understanding how OS interacts with other pathological processes, such as neuroinflammation and metabolic dysregulation, could lead to the development of novel antioxidant compounds that are more effective in treating DN

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GLP-1 receptors have additionally been identified in dermal fibroblasts and keratinocytes, meaning the drug acts directly on the biology of cells responsible for skin architecture, barrier function, and renewal