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This transition involves upregulation of markers such as CD206, Arg1, and IL-10, enhanced efferocytosis of apoptotic cells, and secretion of growth factors like VEGF, TGF-, and FGF2, which promote angiogenesis, fibroblast activation, and ECM remodeling [41]

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Targeting M2phenotypic TAMs reduces the polarization of macrophages toward the M2 phenotype and raises the amount of M1-TAMs, which in turn strengthens the anti-tumor immune microenvironment (38)