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In NAFLD, hepatic uptake and de novo lipogenesis are increased, while a compensatory enhancement of fatty acid oxidation is insufficient in normalizing lipid levels and may even promote cellular damage and disease progression by inducing oxidative stress, especially with compromised mitochondrial function and increased oxidation in peroxisomes and cytochromes
This transformative journey is facilitated by real-time behavioral adjustments alongside the medication's effects
Last J, Perrech M, Denizci C, Dorn F, Kessler J, Seibl-Leven M
