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Exploring glucagon-like peptide-1 receptor agonists as potential disease-modifying agent in psychiatric and neurodevelopmental conditions: evidence from a drug target Mendelian randomization

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Much evidence suggests that the deficiency of this hormone increases insulin resistance, impairs lipid metabolism, augments oxidative damage and deregulates the neuro-hormonal axis

This blockade inhibits the transcription of estrogen-responsive genes that drive cell proliferation, thereby suppressing tumor growth

Optogenetics to causally manipulate a neuronal population and test whether a nootropic peptide acts via this circuit

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