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In contrast, HGF-independent activation occurs when the receptor bypasses the need for its ligand entirely through MET gene amplification or constitutive mutations that force the kinase into an active configuration [6]

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Fish Shellfish Immunol 49:427435 Stec DE, Gordon DM, Hipp JA, Hong S, Mitchell ZL, Franco NR et al (2019) Loss of hepatic PPAR promotes inflammation and serum hyperlipidemia in diet-induced obesity

Hinge like the angiotensin IV antagonist Norleual, was established as a c-Met antagonist by its ability to block HGF-dependent c-Met phosphorylation and prevent HGF-dependent scattering in the MDCK epithelial cell line

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