GSH can also modulate the activity of ion channels, transporters, and enzymes, including glutamate transporters, voltage-gated ion channels, and nitric oxide synthase, through redox-sensitive mechanisms
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S.BlagboroughA
Although some studies confirm the feasibility of exempting patients who achieve pCR after neoadjuvant systemic therapy under rigorous evaluation using ultrasound or MRI, 323,326,327 this approach is constrained by regional development levels and technological disparities
Common clinical manifestations of malonyl CoA deficiency include psychomotor retardation, seizures, hypotonia, hypertrophic cardiomyopathy, metabolic acidosis, diarrhea, vomiting, and hypoglycemia