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Adenovirus-mediated overexpression of CD36 enhances hepatic fatty acid uptake and fat accumulation [22], while liver-specific knockout of CD36 decreases hepatic lipid levels in both genetic and diet-induced steatosis [23]
Compelling evidence suggests that mitochondria are downstream effectors of the death receptors-mediated extrinsic apoptotic pathway via t-Bid-mediated MOMP [83]
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Ongoing follow-up every 1 to 3 months, including weight, blood pressure, heart rate, side effects, and adjustments to nutrition and activity plans
