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Consistent with these findings, supplementation of only GSH/GSHee, but not other antioxidants (Trolox, Ferrostatin-1) prevented induction in SLC25A39 protein levels upon GSH depletion (Fig

In line with the intracellular contributions to these clinical findings, correlation of intraepithelial cell GSH levels to FEV 1 was considerably stronger than the relationship in BAL fluid, supporting the overall importance of endogenous epithelial cell responses to clinical outcomes

The two cell lines exhibited similar behavior and 50 M curcumin was enough to efficiently counteract iron overload toxicity (Fig

The majority of glycine in the body is used for protein synthesis

Structural insights into GHK-Cu stability

Oishi, 2009)