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This study also revealed Cdk5 as an upstream regulator of mitochondrial dysfunction

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This binding triggers a cAMP-mediated signaling cascade that suppresses the transcription of multiple pro-inflammatory genes

When the body is trapped in the vicious cycle of oxidative stress and mitochondrial degradation, targeted intervention is required to break the loop

Therefore, together these studies imply that mutant proteins linked to ALS induce damage to mitochondrial structure, impair its function, dysregulate energy metabolism, and disturb redox homeostasis