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Based on recent studies, the functional and phenotypic changes in smooth muscle cells play a vital role in the onset and progression of atherosclerosis (Figure 5), but the specific mechanism by which smoking regulates smooth muscle cells still requires further research

Studies have shown a significant increase in the levels of autophagic proteins in the lung tissues of patients with COPD at different stages of disease ( in vivo inflammation, cell dysfunction, and apoptosis observed with chronic exposure to CS (Nozik-Grayck et al., 2008)

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