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Smoking is associated with increased risk of myeloproliferative neoplasms: a general population-based cohort study

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Initial observations described no differences in endothelial-dependent dilation between habitual users of combustible tobacco products, users of electronic cigarettes, dual users, and non-users [116, 120], although lower endothelial cell nitric oxide production has been identified in users of ENDS and users of combustible tobacco products when compared to non-users [120]