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Longer exposures of 2-month-old rats or 3-month-old APP/PS1 transgenic mice to sidestream cigarette smoke (1h/day x 5 days/wk x two or 4 months) induces tau and amyloid pathology like that reported in MCI and patients with dementia ( i.e ., insulin signaling, oxidative stress) and the accumulation of neuropathological proteins ( i.e ., tau, amyloid)

This is a big percentage difference however there are also other factors that can be considered that may affect the drop-in success rate

The more someone smokes, and the higher the frequency of smoking, the longer nicotine takes to leave the body

Exposure to secondhand smoke irritates the airways and has immediate harmful effects on a persons heart and blood vessels

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