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However, vast intra- and interfamilial variability tells a much wider clinical presentation [4,5,6] and an important overlap with other EDS subtypes and HCTDs [1, 9, 16, 28,29,30]

These components can induce inflammation in the lungs and airways which leads to damage such as: Narrowing of air passages, Swelling Weakening of the lungs' immunity (increasing susceptibility to infections), Hardening of the inner lung lining Destruction of air sacs The sustained inflammation often persists even after quitting smoking, as the local inflammation remains active

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